Glyphosate linked to non-alcoholic liver steatosis according to study

A new study once again highlights the negative effects of glyphosate, the herbicide and crop dehydrating agents used in particular to kill weeds and in general all those herbs that compete with the crops themselves. It works by inhibiting a particular plant enzyme involved in the synthesis of three aromatic amino acids: tyrosine, tryptophan and phenylalanine. For this reason, it is useful only for plants that are growing and not before germination.

According to the new study, published in Clinical Gastroenterology and Hepatology, glyphosate would be related to non-alcoholic fatty liver disease. Researchers at the University of California examined urine samples from two groups of patients, one diagnosed with NASH (non-alcoholic steatohepatitis, a type of non-alcoholic liver steatosis or NAFLD) and one without. The results, according to the researchers, were quite significant: traces of glyphosate were significantly more present in patients with NASH than in healthy patients, regardless of other factors such as age, race, body mass index and presence of diabetes.

These results, together with other previous research that had been carried out on animals, show that the increase in non-alcoholic liver steatosis in the United States is linked to the increased use of commercial glyphosate.

The tones of Paul J. Mills, Professor of Medicine at the University of San Diego and lead author of the research, appear among other things quite alarming in general terms: “There are so many synthetic chemicals to which we are regularly exposed. We only measured one of them.”

Clive Sullivan

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